(The following contents are copyright © 2001-2004 by Heinrich Kremer)
No reprinting without express permission.
THE SILENT REVOLUTION
IN CANCER AND AIDS MEDICINE
New fundamental knowledge regarding the actual causes of illness and death
confirms the effectiveness of biological compensation therapy.
About this book:
The author, Dr. med. Heinrich Kremer, an experienced physician and clinical researcher, describes here worldwide for the first time how and why a cell becomes cancerous, what the real causes of the so-called AIDS disease are, and that cancer and AIDS are the result of an energetic exhaustion of the immune cells. Over 20 years of meticulous research work stand behind these findings, comprised of the most important scientific publications of orthodox medicine in the fields of immunology and cellular biochemistry since the end of WWII.
With this book, Dr. Kremer points out the absurdity of the HIV hypothesis, grounding his skepticism with rigorous science. However, the fact that this work thoroughly refutes the prevailing scholarly opinion that AIDS is caused by a mysterious virus—which to date nobody has been able to conclusively isolate—is but one of many of the book’s aspects. More essential is that Dr. Kremer presents a consistent therapeutic concept of Evolutionary-Biological medicine, for the first time substantiating that indeed all forms of cancer and even AIDS, can not only be stabilized but also healed.
ISBN 3-934196-13-6
(German 2nd Edition available at Ehlers-Verlag GmbH)
Foreword
The knowledge that this book conveys will revolutionize cancer and AIDS therapy in the coming years. Because after having read this book, no responsible doctor will continue to provide such harmful therapy to the patients in his care and trust. This book will inform them about the fatal mistakes of their previous therapies, of which until now they were the unwitting victims.
In addition, the reading of this book is indispensable for the patient afflicted with cancer or AIDS. Herein for the first time, the exact reasons are revealed to the world why neither cancer nor AIDS must inevitably result in death. These two illnesses are the natural result of a systemic imbalance, which cannot only be halted, but also can be healed.
Dedicated to the memory of my teacher and friend:
Prof. Dr. med. Alfred HÄSSIG (1921–1999)
As a long-standing head of the central laboratory of the Swiss Red Cross, Professor of Immunology at Berne, Advisor to the World Health Organization in all continents, President of the International Society for Blood Transfusion, and Chairman of the Study Group for Diet and Immunity, Alfred Hässig was an essential pioneer in the field of hematology, immunology and stress-medicine.
With an exemplary medical ethic, he has tirelessly and courageously made clear the uncertainty of the so-called HIV test and the fatal consequences of toxic AIDS and cancer therapy. In spite of legal persecution up until his death, he imparted the practical alternatives of biological regulation therapy.
Patients around the world will owe their survival to the doctor and researcher Alfred Hässig, who has liberated them from the fatal mistakes of HIV/AIDS medicine. His impressive reminder—that the service to health must always take priority over profit from illness—remains a lasting legacy and lesson, not only for his friends and colleagues but also for his opponents, who are as skilled and resourceful as they are stubbornly misinformed.
Table Of Contents
| Chapter 1: A Disastrously Wrong Decision | (pp. 11 – 22) |
20 years of abusing nitric gases for sexual enhancement — the seemingly puzzling consequences |
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| Chapter 2: The Sensational Discovery | (pp. 23 – 36) |
Gaseous nitrogen monoxide as a bioenergetic regulat or within and between living cells — the gas war between humans and microbes |
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| Chapter 3: The AIDS Puzzle | (pp. 37 – 51) |
Why the AIDS diseases were misinterpreted — inhibition of the gaseous defense is the cause of acquired immune cell weakness |
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| Chapter 4: AIDS is not a Communicable Disease | (pp. 53 – 99) |
Opportunistic infections and Kaposi’s sarcoma were well known long before the AIDS era — a variety of causes trigger the same immune response, as programmed by biological evolution. |
|
| Chapter 5: Challenging the Previously Valid Immune Theories | (pp. 101 – 134) |
How acquired immune cell impairment actually develops |
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| Chapter 6: The Evolutionary Success of our Ancestral Fusion | (pp. 135 – 168) |
How the micro-Gaian milieu functions — the vital role of the mitochondria |
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| Chapter 6: The Evolutionary Success of our Ancestral Fusion | (pp. 135 – 168) |
How the micro-Gaian milieu functions — the vital role of the mitochondria |
|
| Chapter 7: Collective Tunnel Vision | (pp. 169 – 207) |
Why “HIV characteristics” are the outcome of evolutionary biological programming, and are not specific causes of strong and/or continuous immune stress — what the “HIV test” really measures. |
|
| Chapter 8: The Solution to the Cancer Puzzle | (pp. 209 – 246) |
Why normal cells become cancerous - the degeneration of cancer cells into an embryonic state is programmed by evolutionary biology, and is the result of mitochondrial inactivation. |
|
| Chapter 9: HIV/AIDS Medicine Run Amok | (pp. 247 – 289) |
Why AIDS drugs cause cancer, degenerative changes in muscular and nervous cells, and even AIDS itself — the explanation of how AZT, Bactrim/Septra, and their ilk actually work. |
|
| Chapter 10: The Tremendous Task of Reconsideration | (pp. 291 – 379) |
The elementary malpractices of AIDS and cancer medicine - why patients die by chemotherapeutic poisoning |
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| Chapter 11: The Lifesaving Knowledge on Healing | (pp. 381 – 443) |
On the practice of diagnosing, preventing, and treating AIDS, cancer, and other systemic diseases — rebalancing instead of eradication |
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| Chapter 12: The Resistance Against Mass Poisoning in Africa | (pp. 445 – 486) |
The international initiative of President Mbeki — answers from the South African government’s open discussion: on the causes of AIDS in the West and developing nations, on the nontoxic prevention and therapy for AIDS, on AZT’s true mechanism of action, and the global terror epidemic spread by physicians and the media — the international HIV cartel’s refusal to join the discussion, and the disinformation campaign it launched. |
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Chapter 1: A Disastrously Wrong Decision
(pp. 11 – 22)20 years of abusing nitric gases for sexual enhancement — the seemingly puzzling consequences
Over 140 years ago, nitrites and nitrates were known to affect the circulatory system
It has been known for 50 years that nitrites and nitrates in food, industrial products, and pharmaceuticals, can be transformed into carcinogenic nitrosamine
40 years ago, the lifting of the prescription requirement for the immunotoxic and carcinogenic nitrogen gas led to its epidemic abuse as a drug for sexual enhancement
The desirable effects of nitrite intoxication for homosexuals
20 years later, the first homosexual AIDS patients were chronic nitrite consumers
Background to the “politically correct” misinterpretation of the toxic and pharmacotoxic causes of cancer and immune weakness as a viral infection.
Chapter 2: The Sensational Discovery
(pp. 23 – 36)Gaseous nitrogen monoxide as a bioenergetic regulator within and between living cells — the gas war between humans and microbes
The lengthy and late proof of the existence and function of nitrogen oxides in human blood vessels, immune cells, and nerves
The crucial questions that AIDS researchers did not ask
The discovery of the archaic enzyme used for the biosynthesis of NO gas
The regulation of electron flows and proton gradients (redox equilibrium) by NO and its derivatives
NO gas as soluble and diffusive messaging substance, which does not need target receptors
The NO synthesis enzymes have been evolutionarily conserved
Nitrogen oxides are an age-old communication principle within and between cells, which interact with metal ions and sulfur-hydrogen bonds in order to adjust the redox potentials
NO and its metabolites are indispensable in the restraint or killing of intracellular pathogens
Many non-immune cells also produce cytotoxic NO gas, and are involved in the inhibition of fungi, parasites, and viruses, bacteria, and cancer cells
The explosion of experimental and clinical NO research leads to the revision of disease theories and treatment strategies in many important areas of medicine
Chapter 3: The AIDS Puzzle
(pp. 37 – 51)Why the AIDS diseases were misinterpreted — inhibition of the gaseous defense is the cause of acquired immune cell weakness
The clinical and immunological findings of the first patients in medical history to be diagnosed with AIDS
In 1981, doctors and immunologists did not know that there are both NO-producing and non-NO-producing T-helper immune cells
The evolutionary biological formation of a dual strategy for the human immune defense
Control over the balance between NO gas defense and antibody immunity
Excessive and prolonged NO gas production exhausts the detoxification molecules and shifts the immune balance (AIDS)
The pros and cons of the dual strategy of the immune defense
A rational solution to the apparent AIDS mystery
Chapter 4: AIDS is not a Communicable Disease
(pp. 53 – 99)Opportunistic infections and Kaposi’s sarcoma were well known long before the AIDS era — a variety of causes trigger the same immune response, as programmed by biological evolution.
Pneumocystis carinii pneumonia (PCP) is the most frequent indicator illness in western countries, and was seen more than 60 years ago after the use of sulfonamides on premature infants.
PCP in immunotoxically suppressed patients and those with disturbances of antibody binding formation
Experimentally-induced PCP in chemoimmunosuppressed animals. PCP in the Third World as a result of starvation and malnutrition has been well known for centuries
PCP and Kaposi’s sarcoma (AIDS) were seen in chemoimmunosuppressed organ transplant patients in the 60’s
First International Symposium on AIDS in New York denies the extreme immunotoxic burden of the first AIDS patients and instead postulates a “new illness” by a “new AIDS agent”
The forms of Kaposi’s sarcoma in Africa have been well known for centuries
KS was already clinically defined 130 years ago in Europe by the Hungarian dermatologist Moritz Kaposi
All antibiotics are also immunosuppressive to some degree
The hypotheses to the causes of AIDS and cancer, brought forth at the First International Symposium on AIDS, are in unsolvable contradiction to the clinical reality — a “planned series of human experiments” on AIDS patients is explained as necessary, in order to observe whether cancer develops or progresses after direct pharmaceutical blockade of the cellular immune defense.
In 1983, the virus-cancer researchers arbitrarily called the Kaposi’s sarcoma seen in homosexual nitrite users an “enigma” and “fascinating mystery,” although by 1982 animal experiments had already demonstrated that even short-term nitrite use unleashes immunotoxic and carcinogenic effects
Background of the research politics (1971-1984) leading to the misinterpretation of indiscriminate test tube concotions as “HIV characteristics”
25 years after the one-sided promotion of Retrovirus-Cancer research, the “War on Cancer” is announced to be a failure, after the proof is furnished that in fact only nitrite-inhaling homosexual AIDS patients got sick with Kaposi’s cancer, and after Retrovirus-AIDS researchers declared they were perplexed in solving the AIDS Puzzle
The concept of the non-retroviral disease model
Transplantation AIDS by inhibition of the cytotoxic NO synthesis supports the non-retroviral concept
The counterarguments against NO inhibition as the cause of AIDS, as attempts to salvage the Retrovirus-AIDS theory, are totally illogical
The rush to implicate a sexually transmitted infection
Homosexual men have the highest rates of infectious disease of all other risk groups in North America or Europe
The sole presence of congenital or acquired cellular immune weaknesses is not directly implicated as a cancer cause.
Also the apparent proof of HIV in the T-helper immune cells of AIDS patients, was based on the provocation of cellular products which signify the evolutionary-biologically programmed immune response (experimental laboratory AIDS)
The “HIV antibody” test was calibrated so that a positive result will occur in a person with particularly high levels of polyspecific antibodies (not HIV antibodies) in the blood serum
The “HIV positive” laboratory result is based on circular logic
Also due to the medical knowledge 20 years ago, one could have correctly diagnosed the immunotoxic causes of AIDS without the presumption of a “new agent”
Chapter 5: Challenging the Previously Valid Immune Theories
(pp. 101 – 134)How acquired immune cell impairment actually develops
The discovery of the opposing subtypes of T-helper immune cells (T4 cells Type-I and Type-II = Th1 and Th2)
The DTH skin test used in clinical sepsis research as a diagnostic for the Th1/Th2 immune cell balance
Experimental and clinical evidence for the causal relation between Th1/Th2 immune cell balance, dominance of the Type-II cytokine profile, and anergy of the DTH skin reaction in chronic infections
The immune cell balance of “HIV positives” is already disturbed prior to exhibiting “HIV characteristics”
“HIV characteristics” occur predominantly in Th2 cells associated with Type-II cytokine patterns.
The HIV/AIDS theory is a priori false, because the fundamental self/foreign concept of immunology is incorrect
Unresolved discrepancies in the self/foreign immune theory
The basic rules of inactivity of T and B cells against the body’s own proteins (“self-tolerance”)
T-helper immune cells do not detect “foreign” and “self”, but rather are particularly sensitive to redox state; as such they act as sensor and effector cells, in the regulation and counterregulation of various disturbances of the redox equilibrium
The dilemma of self-defense versus self-preservation
The experimental findings of the feedback system between polarized T-helper cells and the cytokine patterns, as well as the cytotoxic NO synthesis
The clinical consequences of the T-helper cell status, the associated cytokine synthesis and the inhibition of the overproduction of the cytotoxic NO gas with AIDS, sepsis and autoimmune reactions.
The role of Type-II cytokines and self-tolerance of the T- and B-lymph cells in cancerous cellular transformation
The self-tolerance against the anaerobic colony of the intestinal flora as exosymbiotic discharge of the immune balance of the total organism
A successful pregnancy is possible only under the protection of Th2 dominance, with Type-II cytokine status and the inhibition of cytotoxic NO
The force that controls the immune balance
The worldwide increase of allergies, atopy and AIDS are indicators of the civilization-dependent shift of the immune cell balance towards Th2 dominance.
Immune cell imbalance in old age
Independent of the specific causes, all immune weaknesses are governed by the same evolutionary-biological rules
Chapter 6: The Evolutionary Success of our Ancestral Fusion
(pp. 135 – 168)How the micro-Gaian milieu functions — the vital role of the mitochondria
The gigantic energy supply in the respiratory chain of the mitochondria
The coordinated interaction of DNA between the mitochondria and cell nucleus
The Gaian hypothesis and the theory of endosymbiosis
The genetic information inside the cell nucleus of all eukaryotic organisms, humans included, descended from the fusion of the DNA of several single-celled organisms
The evolutionary history of proto-mitochondrial colonization, as bioreactors for all cellular life
The gas-controlled import/export membranes of the mitochondrial colony are reminders of the early development period of the cellular symbiosis
Unexplained for more than 70 years, the Warburg Effect of anaerobic glucose decomposition in cancer cells (fermentative ATP production in the cell plasma without the use of oxygen) also shows up in T-helper immune cells after stimulation during mitosis
Experimental evidence for the physiological toggle-switch of the oxidative energy supply, between cellular respiration in the mitochondria and the predominantly fermentative ATP synthesis inside the cell plasma (Warburg Effect) in the presence of oxygen during the late phases of cell division
The switching between predominantly aerobic glycolysis and OXPHOS, in fetal cells after birth
Pathophysiological (re)fetalization
The system of calcium exchange between the cell plasma and the mitochondrial symbionts regulates numerous energetic and metabolic processes.
The interaction between NO and calcium act to switch the cellular symbiosis between OXPHOS and aerobic glycolysis (Warburg Effect)
The interaction of NO and calcium inside the mitochondria, during programmed cell death (apoptosis) and sudden cell death (necrosis)
The double-edged sword of peroxynitrite in the control of cellular symbiosis
Counterregulation of the cellular symbiosis after stress from NO and oxygen radicals
The activation of prostaglandin synthesis serves to protect the cellular symbiosis, and is evolutionarily conserved
The Bcl-2 protein locks down the mitochondrial membranes and prevents the return from aerobic glycolysis (Warburg Effect) to the OXPHOS phase of cellular symbiosis
The redox control of the cellular symbiosis between NO and its metabolites and the sulfurous non-protein and protein thiols
The immune cell network as a redox-sensitive early warning system for the cellular symbiosis
The gas-controlled micro-Gaian environment of the cellular symbiosis allows room for the thermodynamic range of oscillation between solid and fluid phases
The list of the systemic and chronic diseases caused by functional and structural disturbances of the mitochondria becomes ever longer
AIDS and cancer are not puzzling phenomena, but overdriven counterregulations of the micro-Gaian milieu
Chapter 7: Collective Tunnel Vision
(pp. 169 – 207)Why “HIV characteristics” are the outcome of evolutionary biological programming, and are not specific causes of strong and/or continuous immune stress — what the “HIV test” really measures.
The futile search of the Nobel prize winner Mullis for the original publication showing “HIV is the probable cause of AIDS”
The astonishing confession from the “HIV” discoverer Montagnier, that the self-defined standards for genuine retrovirus isolation were cast to the wind
The financial battle between the Pasteur-Institute in Paris and the National Cancer Institute of the USA, litigation over the patent rights to the “HIV test”
The ramifications of the psychological mass suggestion of a “new plague of sex and blood”
The self-delusions and other mistakes of the “HIV” discoverers and the “disastrous results”
The outbreak of AIDS in 1981 gave the retrovirus establishment, which till then was only an academic flop, an opportunity that transformed into a tragedy of the public health service
The nonspecific “HIV characteristics” are reliably explained by the concept of cellular symbiosis, in compliance with the evolutionary-biological programs that occur following prooxidative stress
The “HIV” test measures an antibody reaction against what one puts into the test substrate: indeterminate proteins, excreted by repeatedly stressed human immune cells
The experimental findings of the Montagnier team as counterevidence against the disease theory that “HIV is the cause of AID and AIDS”
The experimental findings of the Gallo team as counterevidence against the disease theory “HIV is the cause of AID and AIDS”
The system of genetic and supragenetic counterregulation against nitrosative and oxidative stress is proven in all animals
The laboratory trickery behind the mass production of “HIV proteins” needed for assembly of the “HIV test”
“The unseen hand of the market”
Chapter 8: The Solution to the Cancer Puzzle
(pp. 209 – 246)Why normal cells become cancerous - the degeneration of cancer cells into an embryonic state is programmed by evolutionary biology, and is the result of mitochondrial inactivation.
The controversy regarding the Warburg Effect and RNA tumor viruses
The proof of retroviruses in human cancer cells could be never demonstrated
Primarily, the Warburg Effect and cancerous transformation does not depend on genetic defects in the cellular and mitochondrial DNA
The similarity between fetal and cancer cells on the genetic, metabolic and bioenergetic level
The fundamental supposition of the evolutionary biological program of Type-II counterregulation, for the understanding of fetalization and the Warburg Effect
Dissociation of the interaction between different sections of the cellular and mitochondrial genomes
Cancer cells are no longer able to perish (apoptosis or necrosis) from prooxidative stress, since the mitochondrial membranes remain closed after mitosis, due to long-term counterregulation against oxidative and nitrosative stress
The cancerous cell as permanent regression of the cellular symbiosis into the early evolutionary-biological realm of the Archaebacteria and Proteobacteria
Under pathophysiological conditions, the fluctuating bioenergetic rhythm of cellular symbiosis can in two directions be over- or underdriven (Type-I overregulation and following Type-II counterregulation, respectively)
Why, without compensation of the thiol pools, chemotherapeutic agents selectively strengthen the Type-II counterregulation of cancer cells, favoring the formation of resistant metastases
The treatment of organ transplanted patients with Cyclosporin A, an immunosuppressive and carcinogenic mycotoxin, as model of provocation of cancer cell transformation via closing of the mitochondrial membranes.
The striking similarities between the cellular symbiosis behavior of immune and cancer cells, in organ transplant and AIDS patients
The network of energy and information flow is a program that is self-organized by the variable “semiconductor function” of macromolecular protein complexes
During cancerous transformation, the macromolecular semiconductor function of the mitochondrial membranes is invariably inhibited, via forced restriction of the fluidic micro-Gaian gas mixture.
Since isomerase enzymes modulate the semiconductor properties of the macromolecules, the manipulation of the isomerase enzyme Cyclophilin with Cyclosporin A in organ transplants also leads to cancer cell transformation
The successful evolutionary-biological solution—decoupling the genomic sections of the cell nucleus and the mitochondria upon low fluidic level during the phases of physiological cell division—becomes permanently fixed during cancer cell transformation, at the expense of the mitochondrial activity.
Medications with mitochondrial toxicity lead to an inability to utilize oxygen (pseudohypoxia)
The pseudohypoxic state reactivates the evolutionarily conserved biological programs for survival without oxygen
The findings of genetic expression studies with pseudohypoxia are in fundamental contrast to the prevailing theories of carcinogenesis by random mutation
The survival of metastatic and non-metastatic cancer cells does not depend on coincidence, but upon nonlinear bioenergetic conditions
Experimental evidence that programmed cell death in non-metastatic cancer cells depends on activity of the iNOS enzyme for the production of cytotoxic NO gas, and can also be activated in metastatic cancer cells via strong and repeated iNOS stimulation.
In cancer cells, the delicate balance of calcium-dependent NO gases plays a role in the complex Type-II counterregulation and “resistance” after chemotherapy
The causal relationship between chronic fungal and parasitic infections and the development of cancer, after exhaustion of the thiol pool and inhibition of the synthesis of cytotoxic NO
For many decades, nitrosamine researchers sought the primary cause of cancer in the wrong place: in the genome of the cell nucleus
An alternative assertion to the primary cause of cancer, by the concept of the Type-II counterregulation
This alternative concept explains the primary cause of cancer and the relative inefficacy of conventional cancer therapy, substantially more plausibly than any theories of mutation or fictitious retrovirus hypotheses
The drastic decline of infectious and inflammatory processes as major causes of death and disease in industrialized nations, well before introduction of vaccines, chemotherapeutic agents and antibiotics
The dramatically modified nitrosative and oxidative load created in industrialized nations, can be answered by the archaic cellular symbiosis no differently than in the preindustrial evolution phase, and is the cause for the cancer and other chronic degenerative illnesses
All relevant scientific data from 20 years of Kaposi’s sarcoma research, acknowledges excessive nitrosative stress and exhaustion of the thiol pool as the primary cause of the exclusive incidence of KS in homosexual men
In contrast to the Retrovirus-AIDS-Cancer theory, the evolutionary-biological concept of Type-II cellular dysbiosis and cancer cell transformation integrates the entirety of crucial research data without contradiction
Chapter 9: HIV/AIDS Medicine Run Amok
(pp. 247 – 289)Why AIDS drugs cause cancer, degenerative changes in muscular and nervous cells, and even AIDS itself — the explanation of how AZT, Bactrim/Septra, and their ilk actually work.
Three of the most “successful” immune suppressive (azathioprine), antimicrobial (trimethoprim) and “anti-retroviral” (AZT) medicines originate from the same laboratory kitchen: all three substances are immunotoxic, mitochondriotoxic and carcinogenic (primary and secondary AIDS indicator diseases)
Background and manipulations behind the introduction of AZT for planned human experiments
Shedding light on the actual mechanism of AZT
The therapeutic pseudo-benefit of AZT administration
An important question, whether male sperm cells contain immunotoxic and carcinogenic azidothymidine (AZT)
The suppression of toxicological evidence on the carcinogenic properties of AZT by the FDA (the US approval authority), and the worldwide clinical aftermath of AIDS and cancer caused by AZT medication
The manufacturer of AZT confesses that the toxic consequences of AZT administration cannot be differentiated from the theoretical “HIV symptoms”
The working-mechanism of Bactrim/Septra (Trimethioprim+Sulfamethoxazole)
The dangerous illusion of safeguard from infection by continual prophylaxis with Bactrim, in promiscuous homosexuals during the 70’s
After a series of Bactrim-related deaths, it was recommended that its use be restricted, except for in the treatment of already immune-damaged “HIV positives” and AIDS patients
Animal experiments prove the similar-working immunosuppressive effects of Azathioprine and Trimethoprim (Bactrim/Septra)
The medical establishment denies the toxic consequences of its chemotherapeutic “miracle weapons” in order to preserve the status quo
Explanation of the mitochondriotoxic working mechanisms of the components of Bactrim
The strong suspicion of transferring irreparable mitochondrial damage from toxic chemoantibiotics and chemotherapeutic agents, over the maternal germline as an inherited disposition for AIDS, cancer, and other chronic illnesses
The fundamental problem of aggravated Type-II counterregulation and cellular dysbiosis in AIDS and cancer patients, forced by chemoantibiotics and chemotherapeutic agents
The clinical variety of toxic short-term effects following medication with Bactrim/Septra
Proof of the toxic effect of Bactrim/Septra on the DNA
The excessive demand created by Bactrim/Septra on the detoxification performance of the glutathione system, and the incalculable interactions with other chemoantibiotics and chemotherapeutic agents
The question of responsibility for the deadly consequences of “HIV”/AIDS medicine run amok
As a consequence of the rash chemotherapeutic free-for-all, during the 90s “HIV positives” increasingly fell ill to life-threatening bacterial infections not previously seen in these patients
Every antimicrobial substance also attacks human cells, since our own cells bear the heritage of the archaic cellular symbiosis acquired from single-cellular microbes
Chapter 10: The Tremendous Task of Reconsideration
(pp. 291 – 379)The elementary malpractices of AIDS and cancer medicine — why patients die by chemotherapeutic poisoning
The standard publications of “HIV”/AIDS medicine reflect a lack of scientific medical knowledge
The fatal mistake of equating wasting syndrome (cachexia, Gr. kachexein = take away) with a state of chronic hunger
The central role of the exhaustion of the thiol pool (cysteine, glutathione)
The early and late phases of thiol deficiency syndrome
The ornithine-urea cycle of nitrogen transport passes through the mitochondrial cell symbionts and is controlled by cysteine
Why the negative nitrogen balance of wasting syndrome cannot be stopped by “sufficient caloric intake”
The thiol deficiency, and increased use of arginine for urea synthesis and the resultant reduction of NO synthesis, has far-reaching systemic consequences.
Also, the addition of methionine into the protein infusions cannot compensate for the systemic cysteine deficit
With cysteine/glutathione wasting syndrome, the production of the cytotoxic NO gas is inhibited, so that a vicious circle results from the continuous prophylaxis with Bactrim etc., which can lead to the development of counterregulated “resistant” PCP microbes.
Wasting syndrome can be intensified by deficiencies of cysteine and folic acid, as well as many other nutritional deficiencies due to disturbance of the absorption process of the small intestine.
Wasting syndrome develops accordingly with all systemic illnesses e.g. cancer, sepsis, surgical trauma, and ulcerative colitis, among other things.
The thiol deficiency syndrome is associated with all forms of wasting, accordingly associated with cellular immune weakness (AID, pre-AIDS)
Animal experiments have acknowledged the causal relationship between cysteine deficiency, Type-II cytokine status, inhibition of NO synthesis, and wasting syndrome as main cause of death from cancer
Outline of the characteristic laboratory findings with threatening or already occurred wasting syndrome with “HIV positives”, AIDS patients, cancer patients and other systemically gotten sick one
A fundamental understanding that the HIV/AIDS medical profession is missing, is that wasting syndrome (cachexia) has already been clinically defined in connection with characteristic immune weakness, without a primary infectious cause, since the discovery of the Warburg Effect
The fundamental principle of preventive and therapeutic interventions results from the key role of the non-protein thiols
Only a few clinical research groups investigated the most obvious methodology of cysteine compensation therapy via cysteine derivatives
Since AIDS clinicians did not measure the crucial diagnostic parameters, they could not understand the anomalies of cellular immunity in the first AIDS patients
Without the buffer of substantial amounts of cysteine, glutathione, glutamine and arginine, medication with Bactrim, Pentamidine etc. will sooner or later provoke a deadly vicious circle of organ failure, which is explained until today by the laboratory construct of a fictitious immune-weakening virus
The first long-term clinical study on N-Acetyl-Cysteine with “HIV positives” exhibiting strongly degraded values of both T4-helper cell counts and intracellular reduced glutathione (GSH), demonstrates a “dramatically improved probability of survival” despite the senseless counterproductivity of simultaneous administration of the mitochondriotoxic, glutathione-exhausting “combination therapy”
Objections and contradictions between the prominent cysteine research teams
The research-ideological myopia of cysteine-therapy researchers leads to fatal preventive and therapeutic consequences
Since the so-called HIV disease is in reality a mitochondrial illness, it is absurdly untenable to treat it with cysteine compensation and at the same time with nitrosative substances, which are demonstrated to provoke mitochondrial diseases
The vital significance of cysteine compensation is to supply a sufficient amount of freely convertible protons, in order to reverse dysregulation of the switching-rhythm of cellular symbiosis and the resulting negative nitrogenous and energetic balance
The “replenishment of intracellular glutathione” via cysteine supplementation can only occur after a complex and complete changeover, resulting from the increased availability of freely convertible protons
Subtle reductions of reduced glutathione in antigen-presenting cells by toxic substances are sufficient to provoke the so-called “HIV seroconversion,” which in reality is simply a reprogramming of the T4-helper immune cells to the immune cell status of Th2-cells (Type-II cytokine pattern)
The Nobel Prize winner Mullis demands evidence of the disease theory “HIV is the cause of AIDS” that follows the “strict laws of scientific logic”
It is incongruous with the strict laws of scientific logic to warn “HIV positives” against alcohol and acetaminophen as glutathione-exhausting drugs, yet simultaneously conceal the similar-working effects of the chemotherapeutic agents they are so prescribed.
It is incongruous with the strict laws of scientific logic to determine that nitrosative and oxidative substances cause life-threatening damage, even after relatively low dosages if the systemic glutathione levels are previously damaged; and at the same time “preventatively” medicate “HIV positives”, who are pathognomonic for early glutathione insufficiency, with glutathione-exhausting nitrosative and oxidative drugs.
It is incongruous with the strict laws of scientific logic to prescribe nitrosative and glutathione-exhausting chemotherapy to patients who tested “HIV positive” after chronic acetaminophen abuse due to Th1-to-Th2 switch, even though the nitrosative and immunotoxic effects of acetaminophen have been known for over 40 years.
It is incongruous with the strict laws of scientific logic, to on the one hand stimulate via “glutathione replenishment” the proliferation of the Th1-helper immune cells, which in turn allegedly hatch millions of T-cell destructive “HIVs” daily; and on the other hand discover that “HIV positives” provided with “glutathione replenishment” via cysteine supplementation show “dramatically improved survival rates,” as compared to those “HIV positives” that were only treated with the allegedly “HIV-inhibiting cocktail therapy”
It is incongruous with the strict laws of scientific logic to state on the one hand that wasting syndrome (cachexia) is the main cause of death in “HIV positive” AIDS patients (and similarly the main cause of death in patients with cancer and other systemic diseases), and is “a direct consequence of an insufficient amount of oxidative energy metabolism within the mitochondria, as a result of the lowered glutathione level”; and on the other hand prescribe unlimited therapy with glutathione-reducing and mitochondria-inactivating AZT etc. and Bactrim etc., while at best the deadly drama is merely held at bay with brief courses of cysteine supplementation.
The data from the most comprehensive clinical study on AZT and Bactrim therapy (Concorde study) with European “HIV positive” patients proved that administration of AZT and Bactrim could not reduce AIDS incidence and death rates either in early or late stages of illness.
The data from the Concorde study is a compelling argument for the primarily-acquired serious lack of thiol in the “HIV positive” patients, which is further worsened by treatment with AZT, Bactrim, and other glutathione-exhausting, mitochondriotoxic substances up until deadly organ failure occurs.
The results of the Concorde study and other numerous therapy trials prove that with the combination of glutathione-exhausting, mitochondriotoxic pharmaceuticals used in the treatment of HIV/AIDS, just as with cancer chemotherapy, the switching-on process of Type-II counterregulation of the cellular symbiosis is exponentially accelerated.
The archaebacterial subgenome in the cell nucleus functions as the evolutionary-biological memory for the condition of a lack of freely convertible protons in the human cellular symbiosis system
A comparison of the different methodologies of action and thought between pharmatoxic medicine and non-toxic medicine, speaks against the pharmacotoxic method due to the poor survival rates of chemotherapeutically treated “HIV positive” and cancer patients
Soon after the failure of the chemo-cocktails in the early and late treatment of “HIV positives” and AIDS patients, the hunt for the “virus” should have been obligatorily terminated
Background to the abrupt modification of the “HIV” theory, after the disaster of the deadly AZT/Bactrim mass-poisonings of the mid-90’s
The “kitchen sink” model of Dr. Ho as justification, after counting of the alleged “viral load”, to “hit HIV hard and early” with at least three “anti-retroviral” substances (highly active anti-retroviral therapy = HAART, or combination therapy)
The “kitchen sink” model is lacking any logically plausible justification
With the addition of a protease inhibitor, the HAART combination became an enriched substance that was supposed to blunt the imaginary enzyme-scissors of “HIV”, and the involved parties promised the “Lazarus effect” of “eradicating HIV” in three to four years
The “early and hard” bombardment with the toxic combination therapy, irreparably damages the DNA and respiratory chain of the mitochondria; risk of deadly organ failure remains even years later after ending HAART treatment, since damage to mitochondrial DNA is cumulative (chemo late-consequence syndrome)
The extraordinary variety of short- and long-term cellular, organ and metabolic disturbances from combination therapy with protease inhibitors
Almost all of the proven-toxic effects of HAART and protease inhibitors result in mitochondrial dysfunction, and strongly resemble the spectrum of congenital mitochondrial diseases
The quasi-statistical postulations of the “kitchen sink model”, which form the basis of using the allegedly quantitative PCR “viral load” in the individual administration of chemo-cocktails, were disproven by mathematical analyses as objectively incorrect
Practicing physicians and their “HIV patients” let themselves be impressed by pseudo-mathematical values (in log scale) of the laboratory numbers gained by the alleged PCR “viral load” measurement, which cannot rationally justify the guidance of treatment in individual chemotherapy
Apart from the methodological measurement errors of the PCR technique, a reduction of RNA in the blood plasma is not due to inhibition of “HIV” by combination therapy, but on the contrary is from the increased DNA repair set into motion by such damaging therapy; furthermore, an increase of RNA in the blood plasma is not from “HIV resistance” but rather from the disturbed/faulty repair of the DNA defects resulting from combination therapy
The increased serum levels of niacin seen during AIDS progression due to systemic lack of glutathione, cannot be explained by HIV/AIDS research; this increase is also characteristic following cancer chemotherapy, proving that the fluctuating plasma RNA values are due to the DNA-toxic effects of combination therapy
The relative increase of T4 cells in blood serum after combination therapy is also deceiving the physician and patient, because Th1 and Th2 cells are not differentiated in routine laboratory tests; due to maturation-inhibition of the B-cells [in the bone marrow], the Th2 cells cannot find their interaction partners and migrate back into the bloodstream—however the crucial Th1/Th2 immune balance has not actually improved!
In order to prevent death by poisoning from combination therapy, patients and their physicians must learn to understand why and how mitochondrial diseases develop, and how they can be treated by non-toxic compensation therapy
The elegant refutation by orthodox HIV/AIDS researchers of the “kitchen sink model,” the basis for aggressive mitochondriotoxic combination therapy, furnishes proof that the entire construction of the official disease theory that “HIV causes AIDS” is objectively false
The mainstream HIV/AIDS physicians, however, refused to learn to understand the most elementary principals of cellular biology, as the foundation of rationally justified methods of prevention and therapy; ironically they concluded that the patients who deceased (due to primary and secondary mitochondrial inactivation) did not die from too many toxic drugs, but rather from an insufficient prescription of toxic drugs
Background to the empty promise of salvation: to be able to exterminate “HIV” in three to four years via combination therapy
The subsequent appearance of massive mitochondrial damage and metabolic disturbances due to the intensified “planned human poison-experiments”, is answered by the HIV/AIDS medical profession with the absurd new promise: the elimination of “HIV [characteristics] in latently infected cells” would take 10 to 60(!) years of continuous chemotherapeutic poisoning
The results of numerous clinical studies performed in western countries substantiate that on the basis of the objectively culpable non-treatment of the primary cause of their disease, the so-called HIV positive patients paid with their lives for the absolutely senseless aggressive chemotherapy
At the World AIDS Congress of 2000 in South Africa, the US chemotherapists involuntarily declare their clinical bankruptcy
For the army of 10,000 “HIV” specialists, it is forbidden to think that the glutathione level can be systemically balanced with a daily dose of three to eight grams of the natural amino acid cysteine, for six to eight months, and the AIDS mortality rates would become “dramatically lowered”
The hysterization of the AIDS problem (like the demonization of the cancer problem) reflected a foreboding development in modern medicine; the sales volume of expensive chemotherapeutic agents determines the flow of research funds to laboratories, hospital clinics and special practices
The chief instigators of the excessive chemotherapeutic poisoning are likely to be aware of the fact that “HIV characteristics” are nothing more than decay products of the Type-II cytokine shift of T4-helper immune cells in response to glutathione depletion
The statement of the alleged lowering of mortality rates from “HIV infections” is based on dodgy medical assertions that violate of the rules of the Socratic logic, and cannot be accepted as enhanced survival due to combination therapy
The proven correlation between the glutathione level and the incidence of disease in elderly patients, shows that “HIV positives” can be regarded on the cellular-biological level as prematurely-aged patients, due to their preceding long-term prooxidative (nitrosative and oxidative) overload of the cellular symbiosis
As a final irrational evasion from responsibility for the deadly consequences of the objectively false disease theory, the virus hunters at the World AIDS Congress of 2000 called for the use of still more chemotherapeutic agents, in an absurd combination with vaccines against noninfectious human stress proteins, which are decay products and not the cause of systemic disturbances to the cellular symbiosis
The fact that so-called HIV-positive long-term survivors were without exception not treated with AZT etc., Bactrim etc., proves that “HIV” stigmatized patients die as victims of the elementary malpractices of the Retrovirus-AIDS-Cancer medicine
Chapter 11: The Lifesaving Knowledge on Healing
(pp. 381 – 443)An excerpt of this chapter is available for download here: Kremer_Chapter_XI.pdf
On the practice of diagnosing, preventing, and treating AIDS, cancer, and other systemic diseases — rebalancing instead of eradication
The so-called HIV antibody test as nonspecific indicator (not a test for actual HIV antibodies)
The DTH skin reaction as test for immune cell status and indicator of Th2 dominance
The intracellular and plasma levels of reduced glutathione, in connection plasma levels of cysteine, glutamine, arginine, and glutamate, as well as the immune cell status, as indicators for preventive and therapeutic intervention
A proven deficit of reduced glutathione (GSH) calls for mandatory treatment
The independent variable of strong and/or long-lasting prooxidative stress and the dependent variable of glutathione depletion, activate the causal chain of Type-II counterregulation of the cellular symbiosis
The core concept of restoring the redox balance
The glutathione system of the cellular symbiosis must intercept and convert an enormous amount of poison materials and potentially carcinogenic substances, via coupling with cysteine to form the kidney-excreted mercapturic acid
The continual binding of toxic foreign molecules by the glutathione system consumes an enormous quantity of cysteine, which puts the cellular symbioses at risk for systemic glutathione insufficiency syndrome
Despite proof of the effective reduction of “HIV characteristics” by N-Acetyl-Cysteine, “HIV positives” and AIDS patients were not obligatorily treated with the reducing substances cysteine and glutathione, which are “cheaply and easily available, and practically free from any harmful side effects”
In place of the effective, non-toxic compensation therapy, the medical-industrial cartel unlawfully manipulated the fast-track approval of the expensive, highly toxic and carcinogenic AZT chemotherapy for symptomatic and symptom-free “HIV positives”
The sufficient dosage, and the improved bioavailability of cysteine (NAC) and glutathione (GSH) in combination with coenzyme 1 (NAD) and plant polyphenols
In light of the “extremely few attempts at antioxidant therapy”, the “HIV”-believing therapists also criticize that “instead the system of therapeutic clinical trials uses a surplus of cash, resources and time on highly toxic, insignificantly profitable and worthless medicines (AZT etc.)”
The glutathione system has unique properties of quantum physics, not limited antioxidative functions; via the requisite strongly negative redox potential, the disturbance of glutathione equilibrium controls the “weak interactions” of all bioenergetic and biochemical processes of the cellular symbiosis
Crucial questions for the HIV/AIDS medical profession (and likewise for oncologists); why for over 20 years have they actively ignored the available discoveries as to the vital importance of cysteine/glutathione compensation in systemic disease, and instead counterproductively prescribed prooxidative chemo-cocktails?
Finally 20 years after the first AIDS diagnoses, comes the publication of the first successful clinical trial for the treatment of wasting syndrome with high-dose glutamine/cysteine compensation
Experimental and clinical evidence for the systemic interactions between cysteine/glutathione, glutamine, glutamate and arginine, as well as the synthesis of urea and NO in wasting syndrome (Type-II counterregulation of cellular dysbiosis)
The explanation for the favorable therapeutic effects of the high-dose arginine supply against cellular immune weakness and cancer, and the complementary combination with cysteine/glutathione/glutamine compensation
The “confusion at a higher level” of HIV/AIDS and cancer medicine:“At present the intravenous nutritive solutions and most preparations do not contain glutamine for absorption of nutrition via the small intestine, therefore replenishment is not possible”
A comparative analysis of the experimental and clinical research data for the prophylaxis and treatment of wasting syndrome (cachexia) combined with cellular immune weakness by means of prooxidative chemotherapy versus antioxidative supplementation therapy
Consensus, inconsequence, deficits, and suppression of research regarding individual disposition factors in the preventive and therapeutic study of wasting syndrome (cachexia)
The alleged “risk of HIV infection” in the so-called risk groups was falsely and excessively estimated as propaganda, since in addition to the risk-exposure, individual illness-disposing factors are most crucial to the incidence of “HIV characteristics” (similarly so with cancer and many other systemic illnesses)
The variability of blood type, serum proteins, enzyme groups, and tissue-typing cell proteins, cause amongst other things individual disposition factors (genotypic and phenotypic multiformity = polymorphism, Gr. poly = many, morphe = shape)
Various proofs for the exemplary interaction of exposure- and disposition factors with “HIV positive” hemophiliacs, and the refutation of “HIV infection” from cell-free blood clotting proteins
Political hush money for the hemophiliac “victims” and the show trial against transfusion doctors strengthened the suggestive belief in the “deadly HIV mass-contagion” (At the point in time of the 1993-94 chemotherapy disaster, it was scientifically and medically undeniable that the main cause of illness and death for AIDS and wasting syndrome was the standard administration of AZT etc. and Bactrim etc. without therapeutic redox-compensation
The individual disposition for quicker and longer-lasting glutathione depletion explains why in the homosexual and drug injecting risk groups, given the same or higher risk-exposure, only a consistently low percentage of the exposed individuals a develop a strong and/or persistent Type-II cytokine shift (Th1-Th2 switch) that activates the production of higher quantities of polyspecific antibodies (“HIV positive” test result)
The fact that in places with the highest density of drug injection, during 15 years not a single “HIV” seroconversion could be found (in contrast to proven hepatitis B seroconversions), proves the fiction of “HIV transmission” and the individual dependency of disposition factors for the development of a persistent Th2 immune cell dominance (“HIV positive” test result)
The dependency upon disposition for the occurrence of Type-I overregulation and the following Type-II counterregulation is reflected in both the total population and risk groups, as a Gaussian (bell-curve) distribution curve with the vast majority of exposed individuals having a variable immune balance and flexible cytokine pattern; however with chemotherapeutic exposure a generalized weakness of intra- and extracellular immunity emerges
The actual number of reported AIDS cases in Africa is “surprisingly” low, in sharp contrast to the propagandistic numbers of the media and international organizations; disposition factors have not been closely investigated in relation to this population’s generally increased exposure to multiple immune stressors, since these factors are not recorded during African “HIV” testing practices
Over the course of evolution it was favorable to have the individual disposition for an exceptionally redox-sensitive Th2 immune response against invasive extracellular agents, yet in contemporary times it is now a disadvantage; the toxic and pharmacotoxic immune stressors prevalent in modern civilization trigger exactly the same redox-sensitive immune response, and after exhaustion of the redox control system can in such predisposed individuals provoke a persistent Type-II cellular dysbiosis (AIDS, cancer and other systemic illnesses)
Indicative of an individual disposition for redox-efficiency and the detoxification capacity of the cellular symbiosis, patients with manifest systemic illness depend most urgently on replenishment of the cysteine/glutathione systems
The deterministic forecast of an individual’s disposition based on genetic testing of detoxification-enzyme synthesis is to be criticized for many reasons; in reality, the expression of every single gene is redox-dependent, resting primarily on the relative condition of the bioenergetic redox system.
The guiding criteria for diagnosis, amongst those testing “HIV positive”
Preventing and treating systemic illnesses with cysteine/glutathione compensation
Additional glutamine and arginine compensation to prevent or treat wasting syndrome (cachexia)
Additional liver protection, in particular with acute/chronic hepatitis B or autoimmune hepatitis (Oltipraz, nutritive isothiocyanate, polyphenols, glucuronic acid)
Prostaglandin (PGE2) modulation (essential fatty acids: omega-3/omega-6; COX2 inhibitors)
Individually customized micronutrient compensation (vitamins, minerals, trace elements)
Stabilization of the extracellular matrix (polyanions)
Direct activation of the mitochondrial respiration chain (coenzyme Q10; L-carnitine; possibly lipoic acid and thiamine)
The moderation of hypercortisolism (DHEA-S; glucosamineglycanes; heparin; heparinoids; phytotherapeutic complexes: flavonoids + tannins)
The regulation of cytokine chaos in acute full-blown AIDS requires high-dose cysteine/glutathione compensation + DHEA-S + gammaglobulins, in order to curb the antagonism between simultaneous macrophage hyperactivation and the Type-I cytokine inhibition of T-helper immune cells
Overcoming fear and anxiety, and the profound shift in the knowledge of healing from chemoantibiosis to nontoxic [orthomolecular] cellular symbiosis therapy
Chapter 12: The Resistance Against Mass Poisoning in Africa
(pp. 445 – 486)The international initiative of President Mbeki — answers from the South African government’s open discussion: on the causes of AIDS in the West and developing nations, on the nontoxic prevention and therapy for AIDS, on AZT’s true mechanism of action, and the global terror epidemic spread by physicians and the media — the international HIV cartel’s refusal to join the discussion, and the disinformation campaign it launched.
The information from this final chapter is contained in Heinrich Kremer’s “Answers given to Thabo Mbeki,” available here at Alive & Well SF.
Many thanks to Felix de Fries and www.ummafrapp.de!
Bibliography
The bibliography is available in PDF format here: KremerBibliography.pdf